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Conversely, defective autophagy promotes the accumulation of dysfunctional mitochondria and lipid peroxidation products (e.g., MDA and 4-HNE), further amplifying ROS generation and establishing a feed-forward loop (Filomeni et al., 2015)

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We next assessed SNV mutational signatures by computing trinucleotide signature scores and cosine similarity between each tumour–model pair (Methods, ‘Analysis methods: Mutation signature’)

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