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343 The binding of ICs to FcγR drives immune cell clearance of pathogens or target cells via ADCC, with FcγRIIIb expression levels on the surface of neutrophils correlating inversely with cellular ADCC potency
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For example, Δ ptsI mutants show very different phenotypes in BMDMs where no growth occurs compared to the L2 fibroblast plaquing assay where Δ ptsI mutants are indistinguishable from wild type L
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