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Supporting this model, MAG2 overexpression, which enhances uS3 mono-ubiquitination 2 , partially suppressed the increase in the cellular uS10 ubiquitination level by HEL2 overexpression, even upon anisomycin-induced collisions (Fig
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29 Activation of GABA receptors causes opening of chloride channels, which leads to hyperpolarization of the postsynaptic membrane
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