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NO, in addition to its antiproliferative and vasorelaxing roles, has an important role in antagonizing the effects of endothelins, AT-II, and ROS.[35] Reduced NO levels can be attributed to elevated levels of ROS

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We’ve seen what that looks like with compounded GLP-1s and the harm that has come with it — the hospitalizations, contaminated products, and in some cases, death, PCAC cannot allow peptides to follow that same path.” I don't understand how MAHA can scream about "gold standard" clinical trials for vaccines, but push for the approval of synthetic peptides without proper clinical trials