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Semaglutide works by binding the GLP-1 receptor in pancreatic beta cells and hypothalamic appetite centers, where it enhances glucose-dependent insulin secretion, suppresses glucagon, slows gastric emptying, and reduces hunger signals

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This strategy ideally enables receptor-mediated transcytosis (RMT) across the BBB via TfR functionality, while cRGD targets integrins overexpressed on the inflamed BBB and neurovascular units proximal to Aβ aggregates