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Each methylation reaction NNMT performs uses up a molecule of SAM, the cell's primary methyl donor, and converts nicotinamide into a form that can no longer be recycled back into NAD+, the coenzyme central to virtually every energy-producing pathway in the cell
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Moreover, these changes can be attenuated by inhibition of CD38 in the context of chronic HFD consumption and targeted to improve myelin regeneration in the CNS
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