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Beyond the toxic gain-of-function effects of mHTT, HD pathogenesis involves a range of interconnected mechanisms, including mitochondrial dysfunction, transcriptional dysregulation, impaired autophagy, proteostasis failure, excitotoxicity, oxidative stress, and chronic neuroinflammation [38–43]
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To begin with, can biomarkers of ferroptosis in autoimmune diseases be accurately identified with precision and sensitivity
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