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[PubMed] [Google Scholar] 405.Placer Z.A., Cushman L.L., Johnson B.C
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During cancer development, dysregulated ROS levels disrupt redox homeostasis and enhance metastasis by DNA lesions, genetic instability, metabolic transformation, and activation of mitogenic (RAS-RAF-MEK-ERK and PI3K-Akt) signal pathways
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